When the body of missing protein (ascites) 
Ascites is a clinical sign of disease, ie, not a disease in itself, but the sign of another illness. That is, is like saying that an animal has a fever or pain is a sign in itself.
As such sign has a number of differential diagnoses, ie diseases that can cause this. From cardiovascular disease, congestive heart failure right (by multiple causes at once, heartworm disease, pulmonary stenosis, degenerative valve tricuspid valve, etc. ..) liver disease (liver failure, shunt, liver disease secondary to other processes), kidney disease (glomerulonephritis losing proteins, etc ...).
When a patient has ascites, it means that blood vessels is extravasated liquid (this also will be different depending on the associated pathology)
(see our publication on chart diagnosis by type of ascites) blood vessels between Possible causes of extravasation of fluid are the loss of osmotic potential. Osmosis, a physical-chemical phenomenon used in desalination plants, distilleries and of course by the body, basically says that the water will move from the compartment with less solute (substances that are diluted in water) into the compartment with more solutes when it enters them there is a semipermeable membrane (ie, permeable only to water), also passively, ie without wasting energy.
Our semipermeable membrane body are the blood vessels, water is water that contains blood and solutes, the solutes are several good, but the principal solute responsible for the maintenance of the phenomenon of osmosis in the body is albumin, a small protein synthesized by the liver.
When diagnosing ascites due to lack of protein, mainly with sufficient care should check liver function, and if "no training" is renal protein and, if is protein-losing nephropathy. There is also a protein-losing enteropathy should be checked, though usually in this case the digestive animals also show signs outside the own ascites. Note
radiology images, see plenty of liquid content in the abdomen, and a perihilar pulmonary edema pattern extending from the lung, it is indicative that not only is extravasated fluid in the abdomen, but also in the pulmonary blood vessels ( pulmonary edema), pulmonary edema is of course an emergency that requires immediate treatment regardless of ascites, a complication that usually if requires no such urgency.
the blood test also be noted, as the count of albumin is decreased, whereas glomerular filtration suffer molecules such as glucose, is normal, the diagnosis also is supported by a urine analysis in which there was no protein in urine, thus discarding the proteins were excreted by the kidney. Also see on ultrasound ascitic fluid level of intestinal, urinary bladder and kidney failure which is not observed alterations in morphology and echogenicity.
shock therapy for a patient with signs of ascites and pulmonary edema application is based on diuresis (furosemide bolus dose of 1-2 mg / kg) followed by a continuous infusion of furosemide in serum high osmolarity (better physiological) doses of 1 mg / kg hour.
You must be supplemented espirolonactona treatment, rather than as a diuretic as an inhibitor of aldosterone.
You must enter the patient oxygen cage, to maintain tissue oxygenation.
paracentesis in this case is more than indicated, because the ascitic fluid analysis shows that poor amount of protein present, so will not cause a massive loss of proteins by this route. So is extracted ascites content periodically, gradually decreasing the size and abdominal contents.
we ensure proper nutrition of the animal (if it eats its means and in any case or parenteral nutrition via tube feeding), and address potential complications such as secondary infection (with antibiotic coverage).
periodic weight checks (the progressive decrease in weight indicates that the patient is no longer fluid extravasation), improvement of respiratory signs and analytical control (serum protein and urine analysis) are necessary for monitoring the patient .
Alexis J. Santana González. DVM. Col829
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